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Hemodynamic Monitoring: A Crash Course in Swan-Ganz Catheters and Interpreting Waveforms

Understand pulmonary artery catheters, pressure waveforms, and derived hemodynamic parameters. Essential knowledge for managing shock and complex cardiac patients.

VitalJobs Editorial Team
January 24, 20265 min read
hemodynamics
Swan-Ganz
pulmonary artery catheter
cardiac
shock
5 min readUpdated February 4, 2026
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Hemodynamic Monitoring: A Crash Course in Swan-Ganz Catheters and Interpreting Waveforms

The pulmonary artery catheter (PAC), commonly called the Swan-Ganz, remains a valuable tool for managing complex shock states—even as non-invasive alternatives have emerged. Understanding hemodynamic monitoring makes you a more effective critical care provider.

When PACs Are Still Useful

Despite controversy about outcomes, PACs provide uniquely valuable data in:

  • Cardiogenic shock (differentiating causes)
  • Mixed shock states (septic + cardiogenic)
  • Severe pulmonary hypertension
  • Post-cardiac surgery complications
  • Right heart failure assessment
  • Guiding complex fluid management

Anatomy of the PAC

The standard 4-lumen catheter includes:

  • Distal (PA) port: Measures PA pressure, mixed venous blood
  • Proximal (RA) port: Measures RA/CVP, delivers injectate
  • Thermistor: Near tip, measures cardiac output
  • Balloon port: Inflates for PAWP measurement

Normal Pressures: Know Your Numbers

LocationSystolicDiastolicMean
Right Atrium (CVP)--2-6 mmHg
Right Ventricle15-300-8-
Pulmonary Artery15-308-1510-20
PAWP (Wedge)--6-12 mmHg

Reading the Waveforms

RA (CVP) Waveform

Three peaks: a, c, v waves

  • a wave: Atrial contraction (follows P wave)
  • c wave: Tricuspid bulging during isovolumetric contraction
  • v wave: Atrial filling against closed tricuspid

Clinical pearls:

  • Cannon a waves: AV dissociation
  • Giant v waves: Tricuspid regurgitation
  • Absent a waves: Atrial fibrillation

RV Waveform

Steep upstroke, rapid descent

  • Systolic pressure = PA systolic
  • Diastolic approaches zero (unlike PA)

PA Waveform

  • Dicrotic notch marks pulmonic valve closure
  • PA diastolic approximates PAWP in normal conditions

PAWP (Wedge) Waveform

Looks like RA waveform but delayed

  • Represents left atrial pressure
  • a wave follows ECG P wave by ~200ms
  • v wave occurs after T wave

Giant v waves in wedge: Mitral regurgitation, acute MR can have v waves >2x mean PAWP

Obtaining an Accurate Wedge

  1. Inflate balloon slowly until PA waveform dampens to wedge
  2. Confirm wedge pattern (should look atrial, not PA)
  3. Read at end-expiration
  4. Deflate balloon immediately after reading
  5. Never leave balloon inflated—risk of PA rupture

Wedge should be lower than PA diastolic. If wedge > PA diastolic, you're overwedged or have damping issues.

Calculated Hemodynamic Parameters

Cardiac Output (CO) and Index (CI)

  • CO: Liters/minute (normal 4-8 L/min)
  • CI: CO / BSA (normal 2.5-4.0 L/min/m²)
  • Measured by thermodilution or calculated methods

Systemic Vascular Resistance (SVR)

SVR = [(MAP - CVP) / CO] × 80

  • Normal: 800-1200 dynes·sec/cm⁵
  • High SVR: Vasoconstriction (cardiogenic shock, hypovolemia)
  • Low SVR: Vasodilation (septic shock, anaphylaxis)

Pulmonary Vascular Resistance (PVR)

PVR = [(mPAP - PAWP) / CO] × 80

  • Normal: <250 dynes·sec/cm⁵
  • Elevated in pulmonary hypertension, PE, hypoxemia

Stroke Volume (SV) and Index (SVI)

SV = CO / HR

  • Normal SV: 60-100 mL/beat
  • Normal SVI: 33-47 mL/beat/m²

Shock State Differentiation

Cardiogenic Shock

  • CI: ↓ (<2.2)
  • PAWP: ↑ (>18)
  • SVR: ↑
  • Mixed venous O2: ↓

Septic Shock (Warm)

  • CI: ↑ or normal
  • PAWP: Low to normal
  • SVR: ↓↓
  • Mixed venous O2: ↑ (poor extraction)

Hypovolemic Shock

  • CI: ↓
  • PAWP: ↓
  • SVR: ↑
  • Mixed venous O2: ↓

RV Failure

  • CI: ↓
  • CVP: ↑ (CVP > PAWP)
  • PAWP: Low to normal
  • PA pressures may be elevated

Mixed Venous Oxygen Saturation (SvO2)

Obtained from PA port (truly "mixed" venous blood)

SvO2Interpretation
>75%Normal or hyperdynamic state
60-75%Compensated/early shock
40-60%Significant oxygen supply-demand mismatch
<40%Severe shock, imminent arrest

Falling SvO2 = Increased O2 extraction = Inadequate DO2

Troubleshooting Common Problems

Damped Waveform

  • Air bubbles in line
  • Clot at catheter tip
  • Catheter against vessel wall
  • Kinked catheter

Fix: Flush, reposition, aspirate if needed

Can't Wedge

  • Balloon leak (check for blood return)
  • Catheter migration (check position)
  • PA too dilated to occlude

Permanent Wedge (Won't Unwedge)

  • Catheter has migrated too far distally
  • Danger: Risk of PA rupture, infarction
  • Action: Deflate balloon, pull back catheter

Arrhythmias During Insertion

  • PVCs during RV passage are common
  • Usually resolve when catheter enters PA
  • Have lidocaine ready, consider prophylaxis in irritable hearts

Clinical Decision-Making Examples

Case: Hypotension Post-MI

Data: CI 1.8, PAWP 24, SVR 1800

Interpretation: Cardiogenic shock—low output, elevated filling pressures, compensatory vasoconstriction

Action: Inotropes (dobutamine), consider IABP, revascularization if ongoing ischemia

Case: Septic Shock, Not Responding to Fluids

Data: CI 4.5, PAWP 14, SVR 550

Interpretation: Hyperdynamic sepsis with profound vasodilation

Action: More vasopressors (not more fluid), source control

Case: Post-Cardiac Surgery, Low BP

Data: CI 2.0, PAWP 8, CVP 18

Interpretation: CVP > PAWP suggests RV failure—could be RV infarct, PE, or air embolism

Action: Avoid excessive volume, consider pulmonary vasodilators, inotropic support for RV

The Bottom Line

Hemodynamic monitoring isn't about the numbers in isolation—it's about integrating multiple data points with clinical context to guide therapy. A PAC gives you a window into cardiovascular physiology that can be invaluable in complex cases.

Master the waveforms, know the normal values, and always correlate with what you see at the bedside.

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